Uremia model

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Cyclosporin A induces type III collagen interstitial deposition and fibrosis through stimulation of the transforming growth factor β (TGF-β) signaling pathway, and inhibits extracellular matrix (ECM) degradation through modulation of matrix metallopeptidase 9, which results in an extracellular matrix imbalance that leads to increased levels of urea nitrogen (BUN), tubular injury, interstitial inflammation, and fibrosis, which can be observed in model animals.

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