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Model Introduction
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Oral submucosal fibrous degeneration model
Oral submucosal fibrosis (OSF) is a chronic disease characterized by epithelial atrophy, collagen accumulation and microvascular changes. Four major alkaloids have been identified in betel nut: betulin, betulinic acid, betulinic acid, norepinephrine and norepinephrine. Of these, betulin plays the most dominant role in the development of OSF.
Arecoline
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Gingival hyperplasia model
As A highly effective immunosuppressant, cyclosporine A has been widely used to prevent and treat rejection after tissue and organ transplantation. However, when the drug is used clinically, it can produce certain toxic side effects on human body. Since the 1980s, foreign scholars have generally observed that an important side effect of this drug in the oral cavity is drug-induced gingival hyperplasia. The pathological changes of this model are similar to the clinical manifestations of drug-induced gingival hyperplasia in humans, and it is an ideal animal model to study the mechanism of human gingival hyperplasia induced by cyclosporine A.
Cyclosporin A
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Dental fluorosis model
Clinical human dental fluorosis is mainly caused by excessive fluoride intake in the process of tooth mineralization, which leads to enamel mineralization and abnormal development. The model is simple, short in time, low in cost and easy to observe. It is a good animal model for studying the pathogenesis and prevention measures of dental fluorosis.
Sodium Fluoride
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Tinnitus model
Sodium salicylate is a metabolite of aspirin in vivo and is often used to induce tinnitus in animal models in studies of tinnitus pathogenesis. Large doses of sodium salicylate can cause auditory damage to different levels of the auditory system from the cochlea to the central nervous system, especially to the spiral ganglion of the cochlea, resulting in tinnitus, hearing loss and speech cognitive impairment.
Sodium salicylate
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Melasma model
The etiology of melasma is complex, the main cause of chronic diseases are ultraviolet exposure, endocrine disorders, emotional changes and so on. The experimental animal model of melasma was established by using estrogen (progesterone) to attack mice and local exposure to ultraviolet light.
Progesterone + UV exposure
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